None of the patients in the first group enjoined total normalization of hearing. Its improvement by 10-29 dB was documented in 22 (%) patients. In the second group hearing improved by 15-20 dB in 4 (40%) and by 5-10 dB in 6 (60%) patients. In the third group, 14 (%) patients reported improvement of hearing and reduced noise as early as the first puncture. By the time of discharge from the hospital, their hearing improved by 30-40 dB while the 15-20 dB improvement was documented in 2 (%) patients and the 5-10 dB improvement in another 2 (%) patients. The study has demonstrated the high effectiveness of glucocorticoid therapy especially in the case of transtympanic administration of these steroids.
Vestibular paroxysmia is believed to be caused by the neurovascular compression of the cochleovestibular nerve, as it occurs with other neurovascular compression syndromes (. trigeminal neuralgia). The irregular and unpredictable spells are the most disabling aspect of this condition, making some daily activities, like driving, extremely dangerous. In theory, given its pathophysiology, surgical treatment could be considered. Still, due to the substantial surgical risks involved, this approach is reserved for particular cases where pharmacological treatment is not effective or tolerated. Treatment with carbamazepine (Tegretol®) or oxcarbamazepine (Trileptal®), both anticonvulsants primarily used in the treatment of epilepsy, is usually not only effective in small dosages, but is also diagnostic. Vestibular depressants are not effective.